<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2664-3243</journal-id>
<journal-title><![CDATA[Vive Revista de Salud]]></journal-title>
<abbrev-journal-title><![CDATA[Vive Rev. Salud]]></abbrev-journal-title>
<issn>2664-3243</issn>
<publisher>
<publisher-name><![CDATA[CET-BOLIVIA]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2664-32432021000300066</article-id>
<article-id pub-id-type="doi">10.33996/revistavive.v4i12.109</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Vigilancia epidemiológica de Acinetobacter baumannii multirresistente a nivel hospitalario]]></article-title>
<article-title xml:lang="en"><![CDATA[Epidemiological surveillance of multidrug-resistant Acinetobacter baumannii at hospital level]]></article-title>
<article-title xml:lang="pt"><![CDATA[Vigilância epidemiológica do Acinetobacter baumannii multirresistente em nível hospitalar]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Encalada Álvarez]]></surname>
<given-names><![CDATA[Rosa Carolina]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Arteaga Sarmiento]]></surname>
<given-names><![CDATA[Sandra Denisse]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,aff1  ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
</aff>
<aff id="Af2">
<institution><![CDATA[,aff2  ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2021</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2021</year>
</pub-date>
<volume>4</volume>
<numero>12</numero>
<fpage>66</fpage>
<lpage>86</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.org.bo/scielo.php?script=sci_arttext&amp;pid=S2664-32432021000300066&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.bo/scielo.php?script=sci_abstract&amp;pid=S2664-32432021000300066&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.org.bo/scielo.php?script=sci_pdf&amp;pid=S2664-32432021000300066&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La Acinetobacter baumannii se considera como un agente microbiano de gran importancia clínica debido a la resistencia que ha adquirido a los antimicrobianos, esto trae como consecuencias complicaciones al referir una terapia antibiótica, prolongando la estancia en la hospitalización de los pacientes infectados con esta bacteria, causando un alto grado de mortalidad por su elevada capacidad de proliferación en las diferentes áreas hospitalarias.  Objetivo. Describir la epidemiología de los brotes causados por Acinetobacter baumannii en Latinoamérica, así como también los mecanismos de Resistencia de este patógeno causando inconvenientes al momento de emplear los diferentes tratamientos terapéuticos.  Material y métodos.  Es una revisión sistemática bajo la declaración PRISMA de las investigaciones relacionadas al tema. Para la búsqueda de información se emplearon fuentes de datos provenientes de: Scielo, Redalyc, Google académico, se encontraron 43 artículos de los cuales solo 25 fueron válidos para la investigación.  Resultados. La resistencia que presenta la Acinetobacter baumannii a los antibióticos se incrementó con el pasar de los años. Este incremento de la resistencia se debe a diversos factores entre los cuales se destacan el desarrollo de &#946;-lactamasas dirigidas contra los betalactámicos, ya sea de amplio espectro o carbapenemasas; variaciones en las proteínas que forman parte de la membrana externa bacteriana, en las bombas de expulsión, perdida de porinas, modificaciones del lugar (blanco o diana) de acción de los antibióticos y variaciones en la expresión de proteínas fijadoras de penicilina. Esta variabilidad depende en parte de la capacidad de la bacteria para adquirir genes de resistencia, a menudo a través de una transferencia horizontal de genes  Conclusiones.  La Acinetobacter baumannii se caracteriza por tener múltiples mecanismos de resistencia a antibióticos, lo que ha aumentado las consecuencias nocivas de este potencial patógeno y representa un desafío importante para los pacientes y el personal de salud.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Acinetobacter baumannii is considered as a bacterium of great clinical importance due to the resistance it has acquired to antimicrobials, which has triggered complications when referring an antibiotic therapy, prolonging the stay in the hospital of patients infected with this bacterium, causing a high degree of mortality due to its high proliferation capacity between different hospital areas.  Objective.  To describe the most relevant aspects about the epidemiology of the outbreaks caused by Acinetobacter baumannii in Latin America, as well as the different resistance mechanisms that this pathogen has acquired thus causing inconveniences when using the different therapeutic treatments.  Materials and methods.  a systematic review was carried out, under the PRISMA declaration, for the search of the information were used databases such as: Scielo, Redalyc, Google academic, 43 articles were found of which only 25 were valid for research.  Results.  The resistance of Acitetobacter baumannii to different antibiotic groups has increased over the years. This increase in resistance is due to several factors among which stand out: &#946;-lactamases directed against beta-lactams, either broad spectrum or carbapenemases; variations in proteins of the outer membrane, ejection pumps, loss of porins, modifications of the place (target) of action of the antibiotics and alterations in the expression of penicillin-fixing proteins. This ability depends in part on the ability of this bacterium to acquire resistance genes, often through horizontal gene transfer.  Conclusions.  A. baumannii has developed multiple antibiotic resistance mechanisms, which increase the harmful consequences of this potential pathogen and represents a major challenge for patients and health personnel.]]></p></abstract>
<abstract abstract-type="short" xml:lang="pt"><p><![CDATA[Resumo Acinetobacter baumannii é considerada como uma bactéria de grande importância clínica devido à resistência que adquiriu aos antimicrobianos, o que desencadeou complicações no momento de referir uma terapia antibiótica, prolongando a estadia na hospitalização dos pacientes infectados com esta bactéria, causando um elevado grau de mortalidade devido à sua elevada capacidade de proliferação entre diferentes áreas hospitalares.  Objectivo.  Descrever os aspectos mais relevantes sobre a epidemiologia dos surtos causados por Acinetobacter baumannii a nível da América Latina, bem como os diferentes mecanismos de resistência que adquiriu este patógeno causando com isto inconvenientes o momento de empregar os diferentes tratamentos terapéuticos.  Material e métodos.  Realizou-se uma revisão sistemática, sob a declaração PRISMA, para a busca da informação empregaram-se bases de dados como: Scielo, Redalyc, Google acadêmico, foram encontrados 43 artigos dos quais apenas 25 foram válidos para a pesquisa.  Resultados.  A resistência que apresenta Acitetobacter baumannii aos diferentes grupos de antibioticos aumentou com o passar dos anos. Este aumento da resistência deve-se a diversos factores, entre os quais se destacam: &#946;-lactamases dirigidos contra os beta-lactâmicos, quer de largo espectro quer carbapenemases; variações nas proteínas da membrana externa, as bombas de expulsão, perda de porinas, modificações do local (branco) de acção dos antibioticos e alterações na expressão das proteínas fixadoras de penicilina. Esta capacidade depende em parte da capacidade desta bactéria para adquirir genes de resistência, muitas vezes através da transferência horizontal de genes.  Conclusões:  Acinetobacter baumannii desenvolveu múltiplos mecanismos de resistência aos antibióticos, o que aumentou as consequências nocivas deste potencial patogênico e representa um desafio importante para os pacientes e o pessoal de saúde.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Infecciones por Acinetobacter]]></kwd>
<kwd lng="es"><![CDATA[Acinetobacter baumannii]]></kwd>
<kwd lng="es"><![CDATA[infección hospitalaria]]></kwd>
<kwd lng="es"><![CDATA[Vigilancia epidemiológica]]></kwd>
<kwd lng="es"><![CDATA[Resistencia bacteriana]]></kwd>
<kwd lng="en"><![CDATA[Acinetobacter infections]]></kwd>
<kwd lng="en"><![CDATA[Acinetobacter baumannii]]></kwd>
<kwd lng="en"><![CDATA[hospital infection]]></kwd>
<kwd lng="en"><![CDATA[Epidemiological surveillance]]></kwd>
<kwd lng="en"><![CDATA[Bacterial resistance]]></kwd>
<kwd lng="pt"><![CDATA[Infecções por Acinetobacter]]></kwd>
<kwd lng="pt"><![CDATA[Acinetobacter baumannii]]></kwd>
<kwd lng="pt"><![CDATA[infecção hospitalar]]></kwd>
<kwd lng="pt"><![CDATA[Vigilância epidemiológica]]></kwd>
<kwd lng="pt"><![CDATA[Resistência bacteriana]]></kwd>
</kwd-group>
</article-meta>
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